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dc.contributor.authorTaliyan, Rajeev-
dc.date.accessioned2023-12-13T04:33:04Z-
dc.date.available2023-12-13T04:33:04Z-
dc.date.issued2017-08-
dc.identifier.urihttps://www.sciencedirect.com/science/article/abs/pii/S0006899317302123-
dc.identifier.urihttp://dspace.bits-pilani.ac.in:8080/xmlui/handle/123456789/13386-
dc.description.abstractRemote ischemic post conditioning (RIPOC) has shown to be neuroprotective against cerebral ischemic reperfusion (I/R) injury. However, the RIPOC protection against I/R injury induced cognitive abnormalities still remains elusive. Abundant evidence from earlier studies highlighted the role of heme oxygenase-1 (HO-1) in neuronal survival in various neurodegenerative disorders. Thus, in the present study, we investigated the possible contribution of HO-1 in RIPOC mediated neuroprotection against cerebral I/R injury and associated cognitive deficits.en_US
dc.language.isoenen_US
dc.publisherElsevieren_US
dc.subjectPharmacyen_US
dc.subjectRemote limb ischemic post conditioning (RIPOC)en_US
dc.subjectIschemic reperfusion (I/R)en_US
dc.subjectTumor Necrosis Factor (TNF-α)en_US
dc.titleProtective effect of remote limb post conditioning via upregulation of heme oxygenase-1/BDNF pathway in rat model of cerebral ischemic reperfusion injuryen_US
dc.typeArticleen_US
Appears in Collections:Department of Pharmacy

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