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Please use this identifier to cite or link to this item: http://dspace.bits-pilani.ac.in:8080/jspui/handle/123456789/13398
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dc.contributor.authorTaliyan, Rajeev-
dc.date.accessioned2023-12-13T08:57:30Z-
dc.date.available2023-12-13T08:57:30Z-
dc.date.issued2016-
dc.identifier.urihttps://www.ijp-online.com/article.asp?issn=0253-7613;year=2016;volume=48;issue=1;spage=59;epage=63;aulast=Mittal-
dc.identifier.urihttp://dspace.bits-pilani.ac.in:8080/xmlui/handle/123456789/13398-
dc.description.abstractThe signaling pathways upstream of glycogen synthase kinase-3β (GSK-3β) get reduced during ischemic preconditioning (IPC) in hyperlipidemic rat heart. Pioglitazone, an insulin sensitizer, exerts cardioprotection through GSK-3β. The objective of the study is to investigate the role of pioglitazone on the attenuated cardioprotective effect of IPC in hyperlipidemic rat heart.en_US
dc.language.isoenen_US
dc.publisherMedknowen_US
dc.subjectPharmacyen_US
dc.subjectCreatinine kinaseen_US
dc.subjectGlycogen synthase kinase-3βen_US
dc.subjectIschemia-reperfusionen_US
dc.subjectLactate dehydrogenaseen_US
dc.subjectMyocardial injuryen_US
dc.subjectPioglitazoneen_US
dc.subjectRapamycinen_US
dc.subjectWortmanninen_US
dc.titleEffect of pioglitazone on the abrogated cardioprotective effect of ischemic preconditioning in hyperlipidemic rat hearten_US
dc.typeArticleen_US
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