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Please use this identifier to cite or link to this item: http://dspace.bits-pilani.ac.in:8080/jspui/xmlui/handle/123456789/13420
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dc.contributor.authorTaliyan, Rajeev-
dc.date.accessioned2023-12-14T06:47:06Z-
dc.date.available2023-12-14T06:47:06Z-
dc.date.issued2017-10-
dc.identifier.urihttps://linkinghub.elsevier.com/retrieve/pii/S0022510X17326953-
dc.identifier.urihttp://dspace.bits-pilani.ac.in:8080/xmlui/handle/123456789/13420-
dc.description.abstractInsulin resistance has been reported as a strong risk factor for Alzheimer’s disease (AD). However the molecular mechanisms of association between these still remain elusive. Recently, elevated level of histone deacetylases (HDACs) and reduced histone acetylation has been reported in AD patients.Thus, targeting HDACs could provide a novel therapeutic strategy for treating these co-morbid condition.en_US
dc.language.isoenen_US
dc.publisherElsevieren_US
dc.subjectPharmacyen_US
dc.subjectInsulinen_US
dc.subjectHistone deacetylases (HDACs)en_US
dc.titleHistone deacetylase inhibitor, Valproic acid, ameliorates insulin resistance induced cognitive deficit in miceen_US
dc.typeArticleen_US
Appears in Collections:Department of Pharmacy

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