[P2–130]: AR-A014418, A GSK-3β INHIBITOR, ATTENUATES ALZHEIMER'S- LIKE PATHOLOGY IN HIGH FAT DIET-INDUCED COGNITIVE DEFICIT IN MICE

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DC Field	Value	Language
dc.contributor.author	Taliyan, Rajeev	-
dc.date.accessioned	2023-12-14T06:54:46Z	-
dc.date.available	2023-12-14T06:54:46Z	-
dc.date.issued	2017-07	-
dc.identifier.uri	https://alz-journals.onlinelibrary.wiley.com/doi/full/10.1016/j.jalz.2017.06.780	-
dc.identifier.uri	http://dspace.bits-pilani.ac.in:8080/xmlui/handle/123456789/13421	-
dc.description.abstract	Alzheimer's disease (AD) is characterized by the progressive deposition of fibrillar amyloid-β (Aβ) in the form of extracellular plaques and hyperphosphorylated tau as intraneuronal neuro fibrillary tangles (NFTs). Studies have reported that increased Glycogen synthase kinase-3β (GSK-3β) activity has been associated with hyper phosphorylation of tau. However, the molecular mechanisms of association still remain elusive. Thus, the present study was designed to explore the potential of GSK-3β inhibitor, AR-A014418 in high fat diet (HFD) induced cognitive impairment and AD like pathology.	en_US
dc.language.iso	en	en_US
dc.publisher	Wiley	en_US
dc.subject	Pharmacy	en_US
dc.subject	Alzheimer's disease (AD)	en_US
dc.subject	Glycogen synthase kinase-3β	en_US
dc.subject	High Fat Diet (HFD)	en_US
dc.title	[P2–130]: AR-A014418, A GSK-3β INHIBITOR, ATTENUATES ALZHEIMER'S- LIKE PATHOLOGY IN HIGH FAT DIET-INDUCED COGNITIVE DEFICIT IN MICE	en_US
dc.type	Article	en_US
Appears in Collections:	Department of Pharmacy

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