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Please use this identifier to cite or link to this item: http://dspace.bits-pilani.ac.in:8080/jspui/xmlui/handle/123456789/13449
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dc.contributor.authorGaikwad, Anil Bhanudas-
dc.date.accessioned2023-12-19T09:11:24Z-
dc.date.available2023-12-19T09:11:24Z-
dc.date.issued2010-12-
dc.identifier.urihttps://link.springer.com/article/10.1007/s00467-010-1714-8-
dc.identifier.urihttp://dspace.bits-pilani.ac.in:8080/xmlui/handle/123456789/13449-
dc.description.abstractThe notion that some adult diseases may have their origins in utero has recently captured scientists’ attention. Some of these effects persist across generations and may involve epigenetic mechanisms. Epigenetic modifications, DNA methylation together with covalent modifications of histones, alter chromatin density and accessibility of DNA to cellular machinery, modulating the transcriptional potential of the underlying DNA sequence. Here, we will discuss the different epigenetic modifications and their potential role in and contribution to renal disease developmenten_US
dc.language.isoenen_US
dc.publisherSpringeren_US
dc.subjectPharmacyen_US
dc.subjectDNA metallizationen_US
dc.subjectDiseaseen_US
dc.titleFetal environment, epigenetics, and pediatric renal diseaseen_US
dc.typeArticleen_US
Appears in Collections:Department of Pharmacy

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