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dc.contributor.authorPaul, Atish Tulshiram-
dc.date.accessioned2023-12-29T09:56:16Z-
dc.date.available2023-12-29T09:56:16Z-
dc.date.issued2007-11-
dc.identifier.urihttps://pubs.acs.org/doi/full/10.1021/jm0706923-
dc.identifier.urihttp://dspace.bits-pilani.ac.in:8080/xmlui/handle/123456789/13551-
dc.description.abstractThe target for the anti-inflammatory natural products like amentoflavone (2), which act by interfering with the proinflammatory cytokine pathway (e.g., TNF-α, IL-1β, and NO synthase), is not yet well-defined. Data obtained from docking, electronic, and surface analyses shed some light on steric and electronic complementarity of these molecules to p38 MAPK, thereby suggesting a possible mechanism by which they might reduce the production of proinflammatory cytokines.en_US
dc.language.isoenen_US
dc.publisherACSen_US
dc.subjectPharmacyen_US
dc.subjectInhibitorsen_US
dc.subjectLigandsen_US
dc.subjectMolecular interactionsen_US
dc.subjectPeptides and proteinsen_US
dc.titleEvaluation of Proinflammatory Cytokine Pathway Inhibitors for p38 MAPK Inhibitory Potentialen_US
dc.typeArticleen_US
Appears in Collections:Department of Pharmacy

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