Please use this identifier to cite or link to this item:
http://dspace.bits-pilani.ac.in:8080/jspui/handle/123456789/15359
Full metadata record
DC Field | Value | Language |
---|---|---|
dc.contributor.author | Majumder, Syamantak | - |
dc.contributor.author | Chowdhury, Shibasish | - |
dc.date.accessioned | 2024-08-22T10:37:15Z | - |
dc.date.available | 2024-08-22T10:37:15Z | - |
dc.date.issued | 2023-05 | - |
dc.identifier.uri | URL | - |
dc.identifier.uri | https://www.biorxiv.org/content/10.1101/2023.05.12.540614v2 | - |
dc.identifier.uri | http://dspace.bits-pilani.ac.in:8080/jspui/xmlui/handle/123456789/15359 | - |
dc.description.abstract | Endothelial senescence has been linked to several cardiovascular diseases. Dysregulation of proteins of the shelterin complex including TRF2 and TERF2IP causes senescence as it hampers DNA repair and cell proliferation. However, whether exposure to hyperglycemia interplays with proteins of the shelterin complex thus further dictates the senescent phenotype of endothelial cells (EC) remain to be explored. | en_US |
dc.language.iso | en | en_US |
dc.subject | Biology | en_US |
dc.subject | TERF2IP | en_US |
dc.title | Regulation of shelterin proteins TERF2IP and TRF2 by H3K4me3-p65 axis drives hyperglycemia dependent endothelial senescence | en_US |
dc.type | Plan or blueprint | en_US |
Appears in Collections: | Department of Biological Sciences |
Files in This Item:
There are no files associated with this item.
Items in DSpace are protected by copyright, with all rights reserved, unless otherwise indicated.