Favourable modulation of the inflammatory changes in hypercholesterolemic atherogenesis by a low-molecular-weight heparin derivative

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DC Field	Value	Language
dc.contributor.author	Deepa, P.R.	-
dc.date.accessioned	2021-09-19T16:02:14Z	-
dc.date.available	2021-09-19T16:02:14Z	-
dc.date.issued	2006	-
dc.identifier.uri	https://www.sciencedirect.com/science/article/abs/pii/S016752730500433X?via%3Dihub#aep-abstract-id7	-
dc.identifier.uri	http://dspace.bits-pilani.ac.in:8080/xmlui/handle/123456789/2122	-
dc.description.abstract	In the hypercholesterolemic state, the net result of combined oxidative and nitrosative stress is a pro-inflammatory phenotype that is manifested as increased adhesion molecule expression, enhanced leucocyte trafficking, and increased vascular permeability. The present work explores the inflammatory aspects of hypercholesterolemic atherogenesis, and also evaluates the role of a low-molecular-weight heparin derivative (LMWH), Certoparin, on a biochemical basis.	en_US
dc.language.iso	en	en_US
dc.publisher	Elsiever	en_US
dc.subject	Biology	en_US
dc.subject	Low molecular weight heparin	en_US
dc.subject	Heparin	en_US
dc.subject	Atherogenesis	en_US
dc.subject	Hypercholesterolemia	en_US
dc.subject	Inflammation	en_US
dc.title	Favourable modulation of the inflammatory changes in hypercholesterolemic atherogenesis by a low-molecular-weight heparin derivative	en_US
dc.type	Article	en_US
Appears in Collections:	Department of Biological Sciences

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