dc.contributor.author |
Mahesh, R. |
|
dc.date.accessioned |
2023-11-20T04:03:44Z |
|
dc.date.available |
2023-11-20T04:03:44Z |
|
dc.date.issued |
2014-08 |
|
dc.identifier.uri |
https://link.springer.com/article/10.1007/s00213-014-3705-0 |
|
dc.identifier.uri |
http://dspace.bits-pilani.ac.in:8080/xmlui/handle/123456789/13168 |
|
dc.description.abstract |
Olfactory bulbectomy (OBX) is a widely used model for antidepressant screening and known to induce neurodegeneration in several brain areas. Our earlier studies demonstrated that etazolate produced antidepressant-like effects in behavioral despair models of depression; however, the potential role of etazolate on behavior and morphological changes in the hippocampus region along with its underlying mechanism(s) following OBX has not been adequately addressed. |
en_US |
dc.language.iso |
en |
en_US |
dc.publisher |
Springer |
en_US |
dc.subject |
Olfactory bulbectomy (OBX) |
en_US |
dc.subject |
Phosphodiesterase-4 |
en_US |
dc.subject |
Etazolate |
en_US |
dc.title |
Etazolate, a phosphodiesterase-4 enzyme inhibitor produces antidepressant-like effects by blocking the behavioral, biochemical, neurobiological deficits and histological abnormalities in hippocampus region caused by olfactory bulbectomy |
en_US |
dc.type |
Article |
en_US |