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Chloroquine induces transitory attenuation of proliferation of human lung cancer cells through regulation of mutant P53 and YAP

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dc.contributor.author Chowdhury, Shibasish
dc.contributor.author Chowdhury, Rajdeep
dc.contributor.author Mukherjee, Sudeshna
dc.date.accessioned 2024-08-23T04:17:54Z
dc.date.available 2024-08-23T04:17:54Z
dc.date.issued 2022-11
dc.identifier.uri https://link.springer.com/article/10.1007/s11033-022-08072-y
dc.identifier.uri http://dspace.bits-pilani.ac.in:8080/jspui/xmlui/handle/123456789/15367
dc.description.abstract Non-small cell lung carcinoma (NSCLC) is the most common cause of cancer-associated deaths worldwide. Though recent development in targeted therapy has improved NSCLC prognosis, yet there is an unmet need to identify novel causative factors and appropriate therapeutic regimen against NSCLCs. en_US
dc.language.iso en en_US
dc.publisher Springer en_US
dc.subject Biology en_US
dc.subject Non-small cell lung carcinoma (NSCLC) en_US
dc.subject Mutant P53 en_US
dc.title Chloroquine induces transitory attenuation of proliferation of human lung cancer cells through regulation of mutant P53 and YAP en_US
dc.type Article en_US


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