dc.contributor.author |
Chowdhury, Shibasish |
|
dc.contributor.author |
Chowdhury, Rajdeep |
|
dc.contributor.author |
Mukherjee, Sudeshna |
|
dc.date.accessioned |
2024-08-23T04:17:54Z |
|
dc.date.available |
2024-08-23T04:17:54Z |
|
dc.date.issued |
2022-11 |
|
dc.identifier.uri |
https://link.springer.com/article/10.1007/s11033-022-08072-y |
|
dc.identifier.uri |
http://dspace.bits-pilani.ac.in:8080/jspui/xmlui/handle/123456789/15367 |
|
dc.description.abstract |
Non-small cell lung carcinoma (NSCLC) is the most common cause of cancer-associated deaths worldwide. Though recent development in targeted therapy has improved NSCLC prognosis, yet there is an unmet need to identify novel causative factors and appropriate therapeutic regimen against NSCLCs. |
en_US |
dc.language.iso |
en |
en_US |
dc.publisher |
Springer |
en_US |
dc.subject |
Biology |
en_US |
dc.subject |
Non-small cell lung carcinoma (NSCLC) |
en_US |
dc.subject |
Mutant P53 |
en_US |
dc.title |
Chloroquine induces transitory attenuation of proliferation of human lung cancer cells through regulation of mutant P53 and YAP |
en_US |
dc.type |
Article |
en_US |