dc.contributor.author | Chowdhury, Rajdeep | |
dc.contributor.author | Roy, Aniruddha | |
dc.date.accessioned | 2021-09-27T08:09:16Z | |
dc.date.available | 2021-09-27T08:09:16Z | |
dc.date.issued | 2020 | |
dc.identifier.uri | https://link.springer.com/article/10.1007%2Fs43440-020-00122-1 | |
dc.identifier.uri | http://dspace.bits-pilani.ac.in:8080/xmlui/handle/123456789/2324 | |
dc.description.abstract | Recent studies have demonstrated that autophagy plays a critical role in reducing the drug sensitivity of docetaxel (DTX) therapy. Disulfiram (DSF) has exhibited potent autophagy inducing activity in multiple studies. We hypothesized that DSF co-treatment could sensitize breast cancer cells to DTX therapy via autophagy modulation. | en_US |
dc.language.iso | en | en_US |
dc.publisher | Springer | en_US |
dc.subject | Biology | en_US |
dc.subject | Disulfiram | en_US |
dc.subject | Autophagy | en_US |
dc.subject | Breast Cancer Cell | en_US |
dc.title | Disulfiram potentiates docetaxel cytotoxicity in breast cancer cells through enhanced ROS and autophagy | en_US |
dc.type | Article | en_US |
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