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Thalidomide attenuates nitric oxide-driven angiogenesis by interacting with soluble guanylyl cyclase

Thalidomide attenuates nitric oxide-driven angiogenesis by interacting with soluble guanylyl cyclase

Majumder, Syamantak
URI: https://bpspubs.onlinelibrary.wiley.com/doi/10.1111/j.1476-5381.2009.00446.x
http://dspace.bits-pilani.ac.in:8080/xmlui/handle/123456789/2440
Date: 2009-11-30

Abstract:

Nitric oxide (NO) promotes angiogenesis by activating endothelial cells. Thalidomide arrests angiogenesis by interacting with the NO pathway, but its putative targets are not known. Here, we have attempted to identify these targets

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