Thalidomide attenuates nitric oxide-driven angiogenesis by interacting with soluble guanylyl cyclase
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Date
2009-11-30
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Publisher
Wiley
Abstract
Nitric oxide (NO) promotes angiogenesis by activating endothelial cells. Thalidomide arrests angiogenesis by interacting with the NO pathway, but its putative targets are not known. Here, we have attempted to identify these targets
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Keywords
Biology, Thalidomide, Nitric oxide-driven, Guanylyl cyclase