Chloroquine induces transitory attenuation of proliferation of human lung cancer cells through regulation of mutant P53 and YAP

dc.contributor.authorChowdhury, Shibasish
dc.contributor.authorChowdhury, Rajdeep
dc.contributor.authorMukherjee, Sudeshna
dc.date.accessioned2024-08-23T04:17:54Z
dc.date.available2024-08-23T04:17:54Z
dc.date.issued2022-11
dc.description.abstractNon-small cell lung carcinoma (NSCLC) is the most common cause of cancer-associated deaths worldwide. Though recent development in targeted therapy has improved NSCLC prognosis, yet there is an unmet need to identify novel causative factors and appropriate therapeutic regimen against NSCLCs.en_US
dc.identifier.urihttps://link.springer.com/article/10.1007/s11033-022-08072-y
dc.identifier.urihttp://dspace.bits-pilani.ac.in:8080/jspui/xmlui/handle/123456789/15367
dc.language.isoenen_US
dc.publisherSpringeren_US
dc.subjectBiologyen_US
dc.subjectNon-small cell lung carcinoma (NSCLC)en_US
dc.subjectMutant P53en_US
dc.titleChloroquine induces transitory attenuation of proliferation of human lung cancer cells through regulation of mutant P53 and YAPen_US
dc.typeArticleen_US

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